Lymphangiogenesis and Intralymphatic Tumor Growth Vascular Endothelial Growth Factor C Promotes Tumor

نویسندگان

  • Terhi Karpanen
  • Mikala Egeblad
  • Marika J. Karkkainen
  • Hajime Kubo
  • Seppo Ylä-Herttuala
  • Marja Jäättelä
  • Kari Alitalo
چکیده

Many solid tumors produce vascular endothelial growth factor C (VEGF-C), and its receptor, VEGFR-3, is expressed in tumor blood vessels. To study the role of VEGF-C in tumorigenesis, we implanted MCF-7 human breast carcinoma cells overexpressing recombinant VEGF-C orthotopically into severe combined immunodeficient mice. VEGF-C increased tumor growth, but unlike VEGF, it had little effect on tumor angiogenesis. Instead, VEGF-C strongly promoted the growth of tumor-associated lymphatic vessels, which in the tumor periphery were commonly infiltrated with the tumor cells. These effects of VEGF-C were inhibited by a soluble VEGFR-3 fusion protein. Our data suggest that VEGF-C facilitates tumor metastasis via the lymphatic vessels and that tumor spread can be inhibited by blocking the interaction between VEGF-C and its receptor.

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تاریخ انتشار 2001